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The development of CVD. Clinical studies have shown that atorvastatin reduces LDL-C and total cholesterol by 36-53%. In patients with dysbetalipoproteinemia, atorvastatin reduced the levels of intermediate-density lipoprotein cholesterol. It has also been suggested that atorvastatin can limit the extent of angiogenesis, which can be useful in the treatment of chronic subdural hematoma. Trade Name Avasta Availability Prescription only Generic Atorvastatin Atorvastatin Other Names Atorvastatin, atorvastatina, atorvastatine, atorvastatinum Related Drugs Nexletol, Nexlizet, Zetia, Praluent, Repatha, simvastatin, rosuvastatin, Lipitor, fenofibrate, ezetimibe Type Formula C33H35FN2O5 Weight Average: 558.6398 Monoisotopic: 558.253000445 Protein binding Atorvastatin is highly bound to plasma proteins and over 98% of the administered dose is found in a bound form. Groups Approved Therapeutic Class Manufacturer Available Country Bosnia & Herzegowina Last Updated: January 7, 2025 at 1:49 am Uses Avasta is an HMG-CoA reductase inhibitor used to lower lipid levels and reduce the risk of cardiovascular disease including myocardial infarction and stroke.Avasta is indicated for the treatment of several types of dyslipidemias, including primary hyperlipidemia and mixed dyslipidemia in adults, hypertriglyceridemia, primary dysbetalipoproteinemia, homozygous familial hypercholesterolemia, and heterozygous familial hypercholesterolemia in adolescent patients with failed dietary modifications.Dyslipidemia describes an elevation of plasma cholesterol, triglycerides or both as well as to the presence of low levels of high-density lipoprotein. This condition represents an increased risk for the development of atherosclerosis. Avasta is indicated, in combination with dietary modifications, to prevent cardiovascular events in patients with cardiac risk factors and/or abnormal lipid profiles.Avasta can be used as a preventive agent for myocardial infarction, stroke, revascularization, and angina, in patients without coronary heart disease but with multiple risk factors and in patients with type 2 diabetes without coronary heart disease but multiple risk factors.Avasta may be used as a preventive agent for non-fatal myocardial infarction, fatal and non-fatal stroke, revascularization procedures, hospitalization for congestive heart failure and angina in patients with coronary heart disease.Prescribing of statin medications is considered standard practice following any cardiovascular events and for people with a moderate to high risk of development of CVD. Statin-indicated conditions include diabetes mellitus, clinical atherosclerosis (including myocardial infarction, acute coronary syndromes, stable angina, documented coronary artery disease, stroke, trans ischemic attack (TIA), documented carotid disease, peripheral artery disease, and claudication), abdominal aortic aneurysm, chronic kidney disease, and severely elevated LDL-C levels. Avasta is also used to associated treatment for these conditions: Anginal Pain, Cardiovascular Disease (CVD), Coronary Artery Disease (CAD), Coronary artery thrombosis,

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Dysbetalipoproteinemia, Fredrickson Type III lipidemia, Heterozygous Familial Hypercholesterolemia, High Blood Pressure (Hypertension), High Cholesterol, Homozygous Familial Hypercholesterolemia, Hospitalizations, Hypertriglyceridemias, Mixed Dyslipidemias, Mixed Hyperlipidemia, Myocardial Infarction, Non-familial hypercholesterolemia, Postoperative Thromboembolism, Primary Hypercholesterolemia, Stroke, Transient Ischemic Attack, Elevation of serum triglyceride levels, Heterozygous familial hyperlipidemia, Non-familial hyperlipidemia, Non-fatal myocardial infarction, Primary Hyperlipidemia, Revascularization procedures, Revascularization process, Thrombotic events, Cardiovascular Primary Prevention, Secondary prevention cardiovascular event How Avasta works Avasta is a statin medication and a competitive inhibitor of the enzyme HMG-CoA (3-hydroxy-3-methylglutaryl coenzyme A) reductase, which catalyzes the conversion of HMG-CoA to mevalonate, an early rate-limiting step in cholesterol biosynthesis. Avasta acts primarily in the liver, where decreased hepatic cholesterol concentrations stimulate the upregulation of hepatic low-density lipoprotein (LDL) receptors, which increases hepatic uptake of LDL. Avasta also reduces Very-Low-Density Lipoprotein-Cholesterol (VLDL-C), serum triglycerides (TG) and Intermediate Density Lipoproteins (IDL), as well as the number of apolipoprotein B (apo B) containing particles, but increases High-Density Lipoprotein Cholesterol (HDL-C). In vitro and in vivo animal studies also demonstrate that atorvastatin exerts vasculoprotective effects independent of its lipid-lowering properties, also known as the pleiotropic effects of statins. These effects include improvement in endothelial function, enhanced stability of atherosclerotic plaques, reduced oxidative stress and inflammation, and inhibition of the thrombogenic response. Statins were also found to bind allosterically to β2 integrin function-associated antigen-1 (LFA-1), which plays an essential role in leukocyte trafficking and T cell activation. Toxicity The reported LD50 of oral atorvastatin in mice is higher than 5000 mg/kg. In cases of overdose with atorvastatin, there is reported symptoms of complicated breathing, jaundice, liver damage, dark urine, muscle pain, and seizures. In case of overdose, symptomatic treatment is recommended and due to the high plasma protein binding, hemodialysis is not expected to generate significant improvement.In carcinogenic studies with high doses of atorvastatin, evidence of rhabdomyosarcoma, fibrosarcoma, liver adenoma, and liver carcinoma were observed.In fertility studies with high doses of atorvastatin, there were events of aplasia, aspermia, low testis and epididymal weight, decreased sperm motility, decreased spermatid head concentration and increased abnormal sperm.Avasta was shown to not be mutagenic in diverse mutagenic assays. Food Interaction Avoid grapefruit products. Grapefruit products may increase the risk for atorvastatin related adverse effects such as myopathy and rhabdomyolysis.Take with or without food. Food decreases absorption but not to a clinically significant extent.[Moderate] GENERALLY AVOID: Coadministration with grapefruit juice may increase the plasma concentrations of atorvastatin.The proposed mechanism is inhibition

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L. Elimination Route Avasta presents a dose-dependent and non-linear pharmacokinetic profile. It is very rapidly absorbed after oral administration. After the administration of a dose of 40 mg, its peak plasma concentration of 28 ng/ml is reached 1-2 hours after initial administration with an AUC of about 200 ng∙h/ml. Avasta undergoes extensive first-pass metabolism in the wall of the gut and the liver, resulting in an absolute oral bioavailability of 14%. Plasma atorvastatin concentrations are lower (approximately 30% for Cmax and AUC) following evening drug administration compared with morning. However, LDL-C reduction is the same regardless of the time of day of drug administration.Administration of atorvastatin with food results in prolonged Tmax and a reduction in Cmax and AUC.Breast Cancer Resistance Protein (BCRP) is a membrane-bound protein that plays an important role in the absorption of atorvastatin. Evidence from pharmacogenetic studies of c.421C>A single nucleotide polymorphisms (SNPs) in the gene for BCRP has demonstrated that individuals with the 421AA genotype have reduced functional activity and 1.72-fold higher AUC for atorvastatin compared to study individuals with the control 421CC genotype. This has important implications for the variation in response to the drug in terms of efficacy and toxicity, particularly as the BCRP c.421C>A polymorphism occurs more frequently in Asian populations than in Caucasians. Other statin drugs impacted by this polymorphism include fluvastatin, simvastatin, and rosuvastatin.Genetic differences in the OATP1B1 (organic-anion-transporting polypeptide 1B1) hepatic transporter encoded by the SCLCO1B1 gene (Solute Carrier Organic Anion Transporter family member 1B1) have been shown to impact atorvastatin pharmacokinetics. Evidence from pharmacogenetic studies of the c.521T>C single nucleotide polymorphism (SNP) in the gene encoding OATP1B1 (SLCO1B1) demonstrated that atorvastatin AUC was increased 2.45-fold for individuals homozygous for 521CC compared to homozygous 521TT individuals.[A181493] Other statin drugs impacted by this polymorphism include simvastatin, pitavastatin, rosuvastatin, and pravastatin. Half Life The half-life of atorvastatin is 14 hours while the half-life of its metabolites can reach up to 30 hours. Clearance The registered total plasma clearance of atorvastatin is of 625 ml/min. Elimination Route Avasta and its metabolites are mainly eliminated in the bile without enterohepatic recirculation. The renal elimination of atorvastatin is very minimal and represents less than 1% of the eliminated dose. Innovators Monograph

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Description 【Aluminum Frame】The AVASTA Uranus City Bike series adopts lightweight aluminum frames, which not only ensures the structural strength of the bike, but also brings great convenience to urban commuting. The Uranus series comes in two sizes of frames: 44cm and 50cm.【SHIMANO 24-Speed】The specially tuned SHIMANO shifter and rear derailleur can provide up to 24 levels of speed regulation. In daily urban cycling, it is possible to cope with various road conditions, including climbing uphill and riding downhill, by easily and smoothly shifting gears.【Dual Disc Brake】The dual aluminum disc brake has better stability and shorter braking distance, providing better safety. Compared with steel disc brakes, it has faster heat dissipation efficiency, greatly improving the service life of the disc. Even in rainy and snowy days, the disc brake can provide excellent braking force.【700 x 40C Tires】 The 700x40C KENDA tires with dual walled aluminum rims can easily cope with various dirt and gravel roads in urban environments, and can withstand stronger impact without being easily damaged, ensuring long-term use of the wheels.【Easy To Assemble】The AVASTA Uranus City Bicycle is 85% pre-assembled, and installation can be completed by adding the remaining components using the tool kit attached to the packaging, which takes approximately 20 to 30 Specifications Frame: Sturdy Aluminum Alloy FrameStem: Aluminum Alloy / φ28.6*φ31.8Disc Brake: Aluminum Dual Front & Rear Disc BrakeCrank Set: 42T Chain Wheel / Aluminum CrankLeft Shift/Brake Lever: Shimano ST-EF500-L / 3-SpeedRight Shift/Brake Lever: Shimano ST-EF500-8R / 8-SpeedFront Derailleur: Shimano FD-TY510 / Dual PullRear Derailleur: Shimano RD-TY300-DCassette Sprocket: Shimano CS-HG200-8 / 8-SpeedFront & Rear Hub: Aluminum / 14G*32H / Quick ReleaseWheel: 700C Aluminum Rim / A/VTyre: KENDA 700X40CSeat Tube: Aluminum / Quick ReleaseRear Rack: Aluminum / Foldable / Up to 55 lbs (25 kg) WeightCapacity: KickstandAluminum / 270 mm Empowering Yourself Crafted for Unparalleled Adventures Free Fast Shipping Free shipping to USA Checked and Tuned Each bike is checked & tuned prior to shipment Easy Assembly 85% assembled include all the tools you need Processing TimeMost orders are shipped within 48 hours after the payment is processed. Once your order has shipped, we will provide you

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Avasta (Lipitor®), is a lipid-lowering drug included in the statin class of medications. By inhibiting the endogenous production of cholesterol in the liver, statins lower abnormal cholesterol and lipid levels, and ultimately reduce the risk of cardiovascular disease. More specifically, statin medications competitively inhibit the enzyme hydroxymethylglutaryl-coenzyme A (HMG-CoA) Reductase, which catalyzes the conversion of HMG-CoA to mevalonic acid. This conversion is a critical metabolic reaction involved in the production of several compounds involved in lipid metabolism and transport, including cholesterol, low-density lipoprotein (LDL) (sometimes referred to as "bad cholesterol"), and very-low-density lipoprotein (VLDL). Prescribing statins is considered standard practice for patients following any cardiovascular event, and for people who are at moderate to high risk of developing cardiovascular disease. The evidence supporting statin use, coupled with minimal side effects and long term benefits, has resulted in wide use of this medication in North America. Avasta and other statins including lovastatin, pravastatin, rosuvastatin, fluvastatin, and simvastatin are considered first-line treatment options for dyslipidemia. The increasing use of this class of drugs is largely attributed to the rise in cardiovascular diseases (CVD) (such as heart attack, atherosclerosis, angina, peripheral artery disease, and stroke) in many countries. An elevated cholesterol level (elevated low-density lipoprotein (LDL) levels in particular) is a significant risk factor for the development of CVD. Several landmark studies demonstrate that the use of statins is associated with both a reduction in LDL levels and CVD risk. Statins were shown to reduce the incidences of all-cause mortality, including fatal and non-fatal CVD, as well as the need for surgical revascularization or angioplasty following a heart attack. Some evidence has shown that even for low-risk individuals (wAvasta is an oral antilipemic agent that reversibly inhibits HMG-CoA reductase. It lowers total cholesterol, low-density lipoprotein-cholesterol (LDL-C), apolipoprotein B (apo B), non-high density lipoprotein-cholesterol (non-HDL-C), and triglyceride (TG) plasma concentrations while increasing HDL-C concentrations. High LDL-C, low HDL-C and high TG concentrations in the plasma are associated with increased risk of atherosclerosis and cardiovascular disease. The total cholesterol to HDL-C ratio is a strong predictor of coronary artery disease, and high ratios are associated with a higher risk of disease. Increased levels of HDL-C are associated with lower cardiovascular risk. By decreasing LDL-C and TG and increasing HDL-C, atorvastatin reduces the risk of cardiovascular morbidity and mortality. Elevated cholesterol levels (and high low-density lipoprotein (LDL) levels in particular) are an important risk factor for. Download full version of Serial Activacion Avasta 2025 (crack included) Serial Activacion Avasta 2025 0 serial numbers found on Smart Serials database. RECENTLY ADDED Outbyte Download AVasta full version. Download AVasta - Antivirus Indonesia 1.0 Download AVasta - Antivirus Indonesia 2.0. Diposting oleh Aji Setio Aji di 00.20 Tidak ada

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Of CYP450 3A4-mediated first-pass metabolism in the gut wall by certain compounds present in grapefruit.When a single 40 mg dose of atorvastatin was coadministered with 240 mL of grapefruit juice, atorvastatin peak plasma concentration (Cmax) and systemic exposure (AUC) increased by 16% and 37%, respectively.Greater increases in Cmax (up to 71%) and="750" ml="" 1.2="" liters="" per="" day).Clinically, high levels of HMG-CoA reductase inhibitory activity in plasma is associated with an increased risk of musculoskeletal toxicity.Myopathy manifested as muscle pain andRhabdomyolysis has also occurred rarely, which may be accompanied by acute renal failure secondary to myoglobinuria and may result in death.ADJUST DOSING INTERVAL: Fibres such as oat bran and pectin may diminish the pharmacologic effects of HMG-CoA reductase inhibitors by interfering with their absorption from the gastrointestinal tract.MANAGEMENT: Patients receiving therapy with atorvastatin should limit their consumption of grapefruit juice to no more than 1 liter per day.Patients should be advised to promptly report any unexplained muscle pain, tenderness or weakness, particularly if accompanied by fever, malaise andTherapy should be discontinued if creatine kinase is markedly elevated in the absence of strenuous exercise or if myopathy is otherwise suspected or diagnosed.In addition, patients should either refrain from the use of oat bran and pectin or, if concurrent use cannot be avoided, to separate the administration times by at least 2 to 4 hours. Avasta Alcohol interaction[Moderate] Concomitant use of statin medication with substantial quantities of alcohol may increase the risk of hepatic injury.Transient increases in serum transaminases have been reported with statin use and while these increases generally resolve or improve with continued therapy or a brief interruption in therapy, there have been rare postmarketing reports of fatal and non-fatal hepatic failure in patients taking statins.Patients who consume substantial quantities of alcohol and/or have a history of liver disease may be at increased risk for hepatic injury.Active liver disease or unexplained transaminase elevations are contraindications to statin use.Patients should be counseled to avoid substantial quantities of alcohol in combination with statin medications and clinicians should be aware of the increased risk for hepatotoxicity in these patients. Drug Interaction Unknown: aspirin, aspirin, aspirin, aspirin, ubiquinone, ubiquinone, apixaban, apixaban, omega-3 polyunsaturated fatty acids, omega-3 polyunsaturated fatty acids, metoprolol, metoprolol, metoprolol, metoprolol, cyanocobalamin, cyanocobalamin, ascorbic acid, ascorbic acid, cholecalciferol, cholecalciferol Disease Interaction Major: liver disease, rhabdomyolysisModerate: cognitive impairment, diabetes, renal disease Volume of Distribution The reported volume of distribution of atorvastatin is of 380

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